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hUCB Plasma Therapeutic for Neurodegenerative Diseases

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Summary

The USPTO granted patent US12605409B1 to the University of South Florida covering a method of treating neurodegenerative diseases using plasma derived from human umbilical cord blood (hUCB plasma). The patent discloses that hUCB plasma attenuated hyperactive mitogen responses and reduced caspase 3/7 activity in ALS patient cells, supporting therapeutic efficacy. Assignee: University of South Florida; inventors: Svitlana Garbuzova-Davis, Jared Carl Ehrhart, and Paul R. Sanberg.

Published by USPTO on changeflow.com . Detected, standardized, and enriched by GovPing. Review our methodology and editorial standards .

What changed

The USPTO issued patent US12605409B1 to the University of South Florida, covering a therapeutic method using human umbilical cord blood (hUCB) plasma for treating neurodegenerative disorders including ALS. The patent discloses that hUCB plasma modulated mitogen cell response and reduced caspase activity in ALS patient cells, suggesting efficacy both as a standalone therapeutic and as an additive to stem cell treatments.

Biotechnology and pharmaceutical companies developing cell-based or plasma therapies for neurodegenerative diseases should review this patent when assessing their IP landscape. The patent's broad claims covering hUCB plasma alone or in combination with stem cells may create market exclusivity considerations for competing therapeutic programs in this space.

Archived snapshot

Apr 21, 2026

GovPing captured this document from the original source. If the source has since changed or been removed, this is the text as it existed at that time.

← USPTO Patent Grants

Plasma derived from human umbilical cord blood for the treatment of neurodegenerative disorders

Grant US12605409B1 Kind: B1 Apr 21, 2026

Assignee

University of South Florida

Inventors

Svitlana Garbuzova-Davis, Jared Carl Ehrhart, Paul R. Sanberg

Abstract

A method of treating neurodegenerative diseases using hUCB plasma is presented herein. hUCB plasma attenuated the hyperactive response (Group III) and potentiated the normal response in Group I ALS patients, but did not alter that of the non-responders to PHA (Group II). The elevated activity of caspase 3/7 observed in the MNCs from ALS patients was significantly reduced by hUCB plasma treatment. The ability of hUCB plasma to modulate the mitogen cell response and reduce caspase activity suggest that the use of hUCB plasma alone, or with stem cells, may prove useful as a therapeutic in ALS patients. hUCB plasma was shown to increase therapeutic efficacy of MNCs as well as decrease apoptosis of MNCs. The cytokine profile of hUCB plasma supports its usefulness as a sole therapeutic as well as an additive to MNCs.

CPC Classifications

A61K 35/51 C12N 5/0665 A61P 25/28

Filing Date

2023-04-14

Application No.

18134720

Claims

3

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Classification

Agency
USPTO
Instrument
Notice
Branch
Executive
Legal weight
Binding
Stage
Final
Change scope
Minor

Who this affects

Applies to
Pharmaceutical companies
Industry sector
3254 Pharmaceutical Manufacturing 3254.1 Biotechnology
Activity scope
Patent grant Therapeutic method Biotech IP
Geographic scope
United States US

Taxonomy

Primary area
Intellectual Property
Operational domain
Legal
Topics
Pharmaceuticals Biotechnology

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